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DESCRIBE <http://purl.uniprot.org/SHA-384/B40303F4681F15649DDF354E05598E170FF8A0A9570F3B9CAE0CC3F40CAE6AFDB66194AA5D419DEEE9BC1EEAFBE1B923>
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http://purl.uniprot.org/SHA-384/B40303F4681F15649DDF354E05598E170FF8A0A9570F3B9CAE0CC3F40CAE6AFDB66194AA5D419DEEE9BC1EEAFBE1B923
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/B40303F4681F15649DDF354E05598E170FF8A0A9570F3B9CAE0CC3F40CAE6AFDB66194AA5D419DEEE9BC1EEAFBE1B923
http://www.w3.org/2000/01/rdf-schema#comment
"Data suggest that pathological cardiac hypertrophy involved class I histone deacetylases HDAC1 and HdAC2 tuberous sclerosis complex 2 (TSC2) and mTOR srine-threonine kinases (mTOR)."
xsd:string
http://purl.uniprot.org/uniprot/#_005B9237CDA22A08B37D0E4E3D8429F778B276463F63E3D8C7735ECD880CB6A71156DE7D68785158D7BF22D72AA317C4
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/B40303F4681F15649DDF354E05598E170FF8A0A9570F3B9CAE0CC3F40CAE6AFDB66194AA5D419DEEE9BC1EEAFBE1B923
http://purl.uniprot.org/uniprot/Q58E49
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/B40303F4681F15649DDF354E05598E170FF8A0A9570F3B9CAE0CC3F40CAE6AFDB66194AA5D419DEEE9BC1EEAFBE1B923
http://purl.uniprot.org/uniprot/#_Q58E49-mappedCitation-27048565
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/B40303F4681F15649DDF354E05598E170FF8A0A9570F3B9CAE0CC3F40CAE6AFDB66194AA5D419DEEE9BC1EEAFBE1B923