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DESCRIBE <http://purl.uniprot.org/SHA-384/BA29BB0FDB59E232561907F360E5FDA1436C34533EBDA2893EA8C3E9757F881CC334809C7281F148DE519D2344AEA2CB>
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http://purl.uniprot.org/SHA-384/BA29BB0FDB59E232561907F360E5FDA1436C34533EBDA2893EA8C3E9757F881CC334809C7281F148DE519D2344AEA2CB
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/BA29BB0FDB59E232561907F360E5FDA1436C34533EBDA2893EA8C3E9757F881CC334809C7281F148DE519D2344AEA2CB
http://www.w3.org/2000/01/rdf-schema#comment
"miR-21 could activate AKT/PI3K signaling pathway via inhibition of PTEN. MiR-21 contributes to wound healing via inhibition of PTEN that activated AKT/PI3K signaling pathway to increase DCs."
xsd:string
http://purl.uniprot.org/uniprot/#_ABBDB58304D32A0EB13DB399E6AEC9560A92B2194C106E326EDBBFDDF9F62ECA1624420B7A0A364D515458516DC3EB21
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/BA29BB0FDB59E232561907F360E5FDA1436C34533EBDA2893EA8C3E9757F881CC334809C7281F148DE519D2344AEA2CB
http://purl.uniprot.org/uniprot/O54857
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/BA29BB0FDB59E232561907F360E5FDA1436C34533EBDA2893EA8C3E9757F881CC334809C7281F148DE519D2344AEA2CB
http://purl.uniprot.org/uniprot/#_O54857-mappedCitation-28374893
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/BA29BB0FDB59E232561907F360E5FDA1436C34533EBDA2893EA8C3E9757F881CC334809C7281F148DE519D2344AEA2CB