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DESCRIBE <http://purl.uniprot.org/SHA-384/BAD4ABB15E5A72D864E1380B871E1136706347BD172507DEE221DCC2AACA3EB489A9034E38C127ED1C326463D0899C17>
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http://purl.uniprot.org/SHA-384/BAD4ABB15E5A72D864E1380B871E1136706347BD172507DEE221DCC2AACA3EB489A9034E38C127ED1C326463D0899C17
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/BAD4ABB15E5A72D864E1380B871E1136706347BD172507DEE221DCC2AACA3EB489A9034E38C127ED1C326463D0899C17
http://www.w3.org/2000/01/rdf-schema#comment
"data indicate that the loss of NP1/2 disrupts several aspects of retinogeniculate development including the initial establishment of AMPAR transmission and the subsequent elimination of inappropriate circuit connections"
xsd:string
http://purl.uniprot.org/uniprot/#_DB19A07251A1DAE4A0BA743AC34918B5A5EA4C9339443A678306372CDAB8458ED49DDA8A24CAF7A4C4A82AB5E20E06D3
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/BAD4ABB15E5A72D864E1380B871E1136706347BD172507DEE221DCC2AACA3EB489A9034E38C127ED1C326463D0899C17
http://purl.uniprot.org/uniprot/O70340
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/BAD4ABB15E5A72D864E1380B871E1136706347BD172507DEE221DCC2AACA3EB489A9034E38C127ED1C326463D0899C17
http://purl.uniprot.org/uniprot/#_O70340-mappedCitation-20392962
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/BAD4ABB15E5A72D864E1380B871E1136706347BD172507DEE221DCC2AACA3EB489A9034E38C127ED1C326463D0899C17