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DESCRIBE <http://purl.uniprot.org/SHA-384/BE977C92D173B2BAC89F4968D459DD0924B14E0C86BDBAA013A2A974656E84ECA9F261B006E8F72574EBEC66801A785F>
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http://purl.uniprot.org/SHA-384/BE977C92D173B2BAC89F4968D459DD0924B14E0C86BDBAA013A2A974656E84ECA9F261B006E8F72574EBEC66801A785F
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/BE977C92D173B2BAC89F4968D459DD0924B14E0C86BDBAA013A2A974656E84ECA9F261B006E8F72574EBEC66801A785F
http://www.w3.org/2000/01/rdf-schema#comment
"these findings suggest a critical role for C3a through autocrine/paracrine induction of C3aR in the pathogenesis of cigarette smoke-induced sterile inflammation and provide new therapeutic targets for the treatment of emphysema."
xsd:string
http://purl.uniprot.org/uniprot/#_8CC4C5E1F4701E51933851F019A300DBFAA1FFF9F766EB4B19BBA7E970AB69FEB93DED595C68051C2CBB30898975BD0F
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/BE977C92D173B2BAC89F4968D459DD0924B14E0C86BDBAA013A2A974656E84ECA9F261B006E8F72574EBEC66801A785F
http://purl.uniprot.org/uniprot/Q16581
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/BE977C92D173B2BAC89F4968D459DD0924B14E0C86BDBAA013A2A974656E84ECA9F261B006E8F72574EBEC66801A785F
http://purl.uniprot.org/uniprot/#_Q16581-mappedCitation-25465103
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/BE977C92D173B2BAC89F4968D459DD0924B14E0C86BDBAA013A2A974656E84ECA9F261B006E8F72574EBEC66801A785F