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http://purl.uniprot.org/SHA-384/C5A2684745226D8857A0D7043EBC214DA969C3D4BF959C507104889D856EFD44CD9C7BC7D31F164256CC687B8C80AB5Fhttp://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/C5A2684745226D8857A0D7043EBC214DA969C3D4BF959C507104889D856EFD44CD9C7BC7D31F164256CC687B8C80AB5Fhttp://www.w3.org/2000/01/rdf-schema#comment"The aim of this study was to investigate the role of DIAPH1 in the physiological response to experimental myocardial I/R in mice. After subjecting wild-type mice to experimental I/R myocardial DIAPH1 expression was increased an effect that was echoed following hypoxia/reoxygenation (H/R) in H9C2 and AC16 cells"xsd:string
http://purl.uniprot.org/uniprot/#_4017F9BE2AE265AB4F0683ACBE9E05C0C51ACB5B617E4D458B4A7CBA32577BB3D95D467CEBFBE19AA9EFF05864C830A0http://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/C5A2684745226D8857A0D7043EBC214DA969C3D4BF959C507104889D856EFD44CD9C7BC7D31F164256CC687B8C80AB5F
http://purl.uniprot.org/uniprot/A0A1E1ERW3http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/C5A2684745226D8857A0D7043EBC214DA969C3D4BF959C507104889D856EFD44CD9C7BC7D31F164256CC687B8C80AB5F
http://purl.uniprot.org/uniprot/#_A0A1E1ERW3-mappedCitation-29239839http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/C5A2684745226D8857A0D7043EBC214DA969C3D4BF959C507104889D856EFD44CD9C7BC7D31F164256CC687B8C80AB5F