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DESCRIBE <http://purl.uniprot.org/SHA-384/C5BF29538ECB9141C4D31B4DD73AB67FB3174200ACDB8F30586DA8E5D68CD05BC4E3B8EF5293E9A0885180DD0B5B1361>
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http://purl.uniprot.org/SHA-384/C5BF29538ECB9141C4D31B4DD73AB67FB3174200ACDB8F30586DA8E5D68CD05BC4E3B8EF5293E9A0885180DD0B5B1361
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/C5BF29538ECB9141C4D31B4DD73AB67FB3174200ACDB8F30586DA8E5D68CD05BC4E3B8EF5293E9A0885180DD0B5B1361
http://www.w3.org/2000/01/rdf-schema#comment
"Our results suggest that promoter methylation may be a mechanism for inactivation of GPX3 possibly leading to subsequent carcinogenesis and progression of HCC"
xsd:string
http://purl.uniprot.org/uniprot/#_36A9FD9E379D31E1B1AC94F2AA4BCC997B83B52AA5457C821078DFD507C0D4A5F3ED946AB96244B1DF86CB6E0FEACCEF
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/C5BF29538ECB9141C4D31B4DD73AB67FB3174200ACDB8F30586DA8E5D68CD05BC4E3B8EF5293E9A0885180DD0B5B1361
http://purl.uniprot.org/uniprot/P22352
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/C5BF29538ECB9141C4D31B4DD73AB67FB3174200ACDB8F30586DA8E5D68CD05BC4E3B8EF5293E9A0885180DD0B5B1361
http://purl.uniprot.org/uniprot/#_P22352-mappedCitation-25445749
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/C5BF29538ECB9141C4D31B4DD73AB67FB3174200ACDB8F30586DA8E5D68CD05BC4E3B8EF5293E9A0885180DD0B5B1361