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DESCRIBE <http://purl.uniprot.org/SHA-384/C61A421E8DFE9BD9E74E565F04C8A69B4E7D6AD7A72CE254FA4AA269FFEF500483F01CC8309717221902B0696F1CAA55>
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http://purl.uniprot.org/SHA-384/C61A421E8DFE9BD9E74E565F04C8A69B4E7D6AD7A72CE254FA4AA269FFEF500483F01CC8309717221902B0696F1CAA55
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/C61A421E8DFE9BD9E74E565F04C8A69B4E7D6AD7A72CE254FA4AA269FFEF500483F01CC8309717221902B0696F1CAA55
http://www.w3.org/2000/01/rdf-schema#comment
"Authors demonstrate that genetic silencing of CXCR2 on oligodendroglia did not affect clinical disease neuroinflammation or demyelination yet there was increased remyelination."
xsd:string
http://purl.uniprot.org/uniprot/#_2FBB9B0377141BF226857A1AA33C18F1C9513650DB3F336BE84F0F381658EF81E59153B605002F28D471B6DCF8B9D864
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/C61A421E8DFE9BD9E74E565F04C8A69B4E7D6AD7A72CE254FA4AA269FFEF500483F01CC8309717221902B0696F1CAA55
http://purl.uniprot.org/uniprot/P25025
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/C61A421E8DFE9BD9E74E565F04C8A69B4E7D6AD7A72CE254FA4AA269FFEF500483F01CC8309717221902B0696F1CAA55
http://purl.uniprot.org/uniprot/#_P25025-mappedCitation-31243125
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/C61A421E8DFE9BD9E74E565F04C8A69B4E7D6AD7A72CE254FA4AA269FFEF500483F01CC8309717221902B0696F1CAA55