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DESCRIBE <http://purl.uniprot.org/SHA-384/C84D88F744262DAD0ADF8E4D765C88A286680C2D0D76C43F3EA346F87784DC6E1E8A1EC755C00CC0F416F2324C4C6AF9>
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http://purl.uniprot.org/SHA-384/C84D88F744262DAD0ADF8E4D765C88A286680C2D0D76C43F3EA346F87784DC6E1E8A1EC755C00CC0F416F2324C4C6AF9
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/C84D88F744262DAD0ADF8E4D765C88A286680C2D0D76C43F3EA346F87784DC6E1E8A1EC755C00CC0F416F2324C4C6AF9
http://www.w3.org/2000/01/rdf-schema#comment
"Taken together these findings indicate that PKR functions as an essential modulator in LPS-induced STAT inflammatory signaling events and provides new insight into endotoxin-induced CNS diseases following infection."
xsd:string
http://purl.uniprot.org/uniprot/#_21490B40A49F2DEBD911947674F75284EF12CC502420E0DEFE93A6283AB6A77FBEDC7AD764DDAF4CC5E814E953EDFDAD
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/C84D88F744262DAD0ADF8E4D765C88A286680C2D0D76C43F3EA346F87784DC6E1E8A1EC755C00CC0F416F2324C4C6AF9
http://purl.uniprot.org/uniprot/Q6P6Q7
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/C84D88F744262DAD0ADF8E4D765C88A286680C2D0D76C43F3EA346F87784DC6E1E8A1EC755C00CC0F416F2324C4C6AF9
http://purl.uniprot.org/uniprot/#_Q6P6Q7-mappedCitation-15630703
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/C84D88F744262DAD0ADF8E4D765C88A286680C2D0D76C43F3EA346F87784DC6E1E8A1EC755C00CC0F416F2324C4C6AF9