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DESCRIBE <http://purl.uniprot.org/SHA-384/C8E2C2BEC8E01B42300522DDB9570436294E364732633897FEC9F849C41D3CA8F2A309057F6C728BBCBD11978C77BEC6>
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http://purl.uniprot.org/SHA-384/C8E2C2BEC8E01B42300522DDB9570436294E364732633897FEC9F849C41D3CA8F2A309057F6C728BBCBD11978C77BEC6
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/C8E2C2BEC8E01B42300522DDB9570436294E364732633897FEC9F849C41D3CA8F2A309057F6C728BBCBD11978C77BEC6
http://www.w3.org/2000/01/rdf-schema#comment
"Findings demonstrate that the pro- and anti-inflammatory responses to gAd in innate immune cells are time-dependent and mediated by the activation of the IkappaB/NF-kappaB pathway and IRAK-1 downregulation respectively."
xsd:string
http://purl.uniprot.org/uniprot/#_1DAB431D95AA7BD7B67AA7D68EE73153149BF621501F84209C4930C6CDEF793A4EF2F0037B5A69D6C19902FFA6BC8587
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/C8E2C2BEC8E01B42300522DDB9570436294E364732633897FEC9F849C41D3CA8F2A309057F6C728BBCBD11978C77BEC6
http://purl.uniprot.org/uniprot/Q8BR10
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/C8E2C2BEC8E01B42300522DDB9570436294E364732633897FEC9F849C41D3CA8F2A309057F6C728BBCBD11978C77BEC6
http://purl.uniprot.org/uniprot/#_Q8BR10-mappedCitation-30078230
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/C8E2C2BEC8E01B42300522DDB9570436294E364732633897FEC9F849C41D3CA8F2A309057F6C728BBCBD11978C77BEC6