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DESCRIBE <http://purl.uniprot.org/SHA-384/C96BC3185BBD2DA00F2CBC59B8433A9D6A1A018B1D56A92ADEBAD11D2AE4791FCC3468309B6FD0BDB6FAFDF9F9971AC6>
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http://purl.uniprot.org/SHA-384/C96BC3185BBD2DA00F2CBC59B8433A9D6A1A018B1D56A92ADEBAD11D2AE4791FCC3468309B6FD0BDB6FAFDF9F9971AC6
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/C96BC3185BBD2DA00F2CBC59B8433A9D6A1A018B1D56A92ADEBAD11D2AE4791FCC3468309B6FD0BDB6FAFDF9F9971AC6
http://www.w3.org/2000/01/rdf-schema#comment
"Tumor-infiltrating natural killer and CD4(+) T cells under the influence of cancer cells significantly increase the production of IFNgamma which in turn inhibits TNFSF15 expression in vascular endothelial cells."
xsd:string
http://purl.uniprot.org/uniprot/#_5032628B6EC7F7EFE62459D7A3BEAE509D73DE3B0DB38F1C69A77C3974612DC8D4332A8D47E8160A6A90016EF45E0E52
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/C96BC3185BBD2DA00F2CBC59B8433A9D6A1A018B1D56A92ADEBAD11D2AE4791FCC3468309B6FD0BDB6FAFDF9F9971AC6
http://purl.uniprot.org/uniprot/O95150
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/C96BC3185BBD2DA00F2CBC59B8433A9D6A1A018B1D56A92ADEBAD11D2AE4791FCC3468309B6FD0BDB6FAFDF9F9971AC6
http://purl.uniprot.org/uniprot/#_O95150-mappedCitation-24141405
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/C96BC3185BBD2DA00F2CBC59B8433A9D6A1A018B1D56A92ADEBAD11D2AE4791FCC3468309B6FD0BDB6FAFDF9F9971AC6