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DESCRIBE <http://purl.uniprot.org/SHA-384/CB7A9194B4B3055264EB1DD36224A43D265350EA79BDB1F141E8544C2A37F6B25AE5F7B7B75C958CD29108C347632F82>
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http://purl.uniprot.org/SHA-384/CB7A9194B4B3055264EB1DD36224A43D265350EA79BDB1F141E8544C2A37F6B25AE5F7B7B75C958CD29108C347632F82
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/CB7A9194B4B3055264EB1DD36224A43D265350EA79BDB1F141E8544C2A37F6B25AE5F7B7B75C958CD29108C347632F82
http://www.w3.org/2000/01/rdf-schema#comment
"These results suggest that inactivation of GIT2 function is a required step for induction of cell motility and that GIT2 may be a target of oncogenic signaling pathways that regulate cell migration."
xsd:string
http://purl.uniprot.org/uniprot/#_D93AD2F7657946C2D8163BD3238DA95DF22B2E93BBB9A5643F9103EE1C96A20D383582E1454481BBA40B6B8556412ED6
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/CB7A9194B4B3055264EB1DD36224A43D265350EA79BDB1F141E8544C2A37F6B25AE5F7B7B75C958CD29108C347632F82
http://purl.uniprot.org/uniprot/Q14161
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/CB7A9194B4B3055264EB1DD36224A43D265350EA79BDB1F141E8544C2A37F6B25AE5F7B7B75C958CD29108C347632F82
http://purl.uniprot.org/uniprot/#_Q14161-mappedCitation-16628223
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/CB7A9194B4B3055264EB1DD36224A43D265350EA79BDB1F141E8544C2A37F6B25AE5F7B7B75C958CD29108C347632F82