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http://purl.uniprot.org/SHA-384/CC80F1B50BDCE75FB91AAF466A00481B4672E2A83C01157C2032EB506081C8F75AF49BA058E91BABEE1A3005E1324E56http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/CC80F1B50BDCE75FB91AAF466A00481B4672E2A83C01157C2032EB506081C8F75AF49BA058E91BABEE1A3005E1324E56http://www.w3.org/2000/01/rdf-schema#comment"Study established a cellular model system characterized by a deficiency of SLC37A4 which presents pathological manifestations of GSD Ib in the kidney. Expression analysis in a novel model system supports the hypothesis that renal dysfunction in the GSD Ib is partly due to the ER stress and increased apoptosis."xsd:string
http://purl.uniprot.org/uniprot/#_75BCA84B8005D46E3F0B5127C507913AA2C039999F8C24DC22B2D2D7BC512E2BC6F532E7CEA65905D6786B044923BAD0http://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/CC80F1B50BDCE75FB91AAF466A00481B4672E2A83C01157C2032EB506081C8F75AF49BA058E91BABEE1A3005E1324E56
http://purl.uniprot.org/uniprot/A8K0S7http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/CC80F1B50BDCE75FB91AAF466A00481B4672E2A83C01157C2032EB506081C8F75AF49BA058E91BABEE1A3005E1324E56
http://purl.uniprot.org/uniprot/#_A8K0S7-mappedCitation-30951856http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/CC80F1B50BDCE75FB91AAF466A00481B4672E2A83C01157C2032EB506081C8F75AF49BA058E91BABEE1A3005E1324E56