RDF/XMLNTriplesTurtleShow queryShare
SubjectPredicateObject
http://purl.uniprot.org/SHA-384/CDD4C5FF1F53EF94A68BD35FC10DFB29393C7B38F360ABCB7037F2C8B41F830157163A1E6D795D929265BCF1A1A80C9Dhttp://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/CDD4C5FF1F53EF94A68BD35FC10DFB29393C7B38F360ABCB7037F2C8B41F830157163A1E6D795D929265BCF1A1A80C9Dhttp://www.w3.org/2000/01/rdf-schema#comment"The results of this case study indicated that although FOXL2 402C > G mutation determines the development of granulosa cell tumor PMS2 mutation may be the initial driver of carcinogenesis. Immunohistochemistry-based tumor testing for mismatch repair gene expression may be necessary for granulosa cell tumors to determine their malignant potential or if they are part of Lynch syndrome."xsd:string
http://purl.uniprot.org/uniprot/#_2557FF7D29F50435808F087258E4CD0CD723E350FB35881C5AEADCE13A68B1E344B99FDE587F1938FA0D2B0EEA360F16http://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/CDD4C5FF1F53EF94A68BD35FC10DFB29393C7B38F360ABCB7037F2C8B41F830157163A1E6D795D929265BCF1A1A80C9D
http://purl.uniprot.org/uniprot/C9J167http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/CDD4C5FF1F53EF94A68BD35FC10DFB29393C7B38F360ABCB7037F2C8B41F830157163A1E6D795D929265BCF1A1A80C9D
http://purl.uniprot.org/uniprot/#_C9J167-mappedCitation-28347324http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/CDD4C5FF1F53EF94A68BD35FC10DFB29393C7B38F360ABCB7037F2C8B41F830157163A1E6D795D929265BCF1A1A80C9D