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http://purl.uniprot.org/SHA-384/CE41EF1CC718944683CB52150D43AE5EB10B34238D6850D3470C070BC9F055D09663519C08BA19E8F47C1FDE2A81A41Dhttp://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/CE41EF1CC718944683CB52150D43AE5EB10B34238D6850D3470C070BC9F055D09663519C08BA19E8F47C1FDE2A81A41Dhttp://www.w3.org/2000/01/rdf-schema#comment"The presented data substantiate the hypothesis that claudin-18 is a central barrier-forming component of tight junctions and show that IL-13 downregulates claudin-18. These data also suggest that the loss of claudin-18 is associated with increased sensitization to aeroantigens and airway responsiveness"xsd:string
http://purl.uniprot.org/uniprot/#_38C94F02DA3526506944A1F4C9D1100FFD6E8CD8733C7D1349B92E40F6AC993EC373D80A32BAA424FD02A1818C7E5A67http://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/CE41EF1CC718944683CB52150D43AE5EB10B34238D6850D3470C070BC9F055D09663519C08BA19E8F47C1FDE2A81A41D
http://purl.uniprot.org/uniprot/P56856http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/CE41EF1CC718944683CB52150D43AE5EB10B34238D6850D3470C070BC9F055D09663519C08BA19E8F47C1FDE2A81A41D
http://purl.uniprot.org/uniprot/#_P56856-mappedCitation-27215490http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/CE41EF1CC718944683CB52150D43AE5EB10B34238D6850D3470C070BC9F055D09663519C08BA19E8F47C1FDE2A81A41D