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DESCRIBE <http://purl.uniprot.org/SHA-384/CFAD8300F2868A5EC8CD44D1CE89A990963AEC6ADE3F6FE992123DB8F9D9A0674DD6E9872A3ABD0E299F9B982F3A9157>
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http://purl.uniprot.org/SHA-384/CFAD8300F2868A5EC8CD44D1CE89A990963AEC6ADE3F6FE992123DB8F9D9A0674DD6E9872A3ABD0E299F9B982F3A9157
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/CFAD8300F2868A5EC8CD44D1CE89A990963AEC6ADE3F6FE992123DB8F9D9A0674DD6E9872A3ABD0E299F9B982F3A9157
http://www.w3.org/2000/01/rdf-schema#comment
"IRF-1 promotes LTx I/R injury via hepatocyte IL-15/IL-15Ralpha production and suggest that targeting IRF-1 and IL-15/IL-15Ralpha may be effective in reducing I/R injury associated with LTx."
xsd:string
http://purl.uniprot.org/uniprot/#_DE081B37C92D8D60807028A4EB43D3D14AE5EB30787779E0FC4A6A01BF04889270051BDAD627A67B5D720C55B427BF03
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/CFAD8300F2868A5EC8CD44D1CE89A990963AEC6ADE3F6FE992123DB8F9D9A0674DD6E9872A3ABD0E299F9B982F3A9157
http://purl.uniprot.org/uniprot/A0A0B6XK11
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/CFAD8300F2868A5EC8CD44D1CE89A990963AEC6ADE3F6FE992123DB8F9D9A0674DD6E9872A3ABD0E299F9B982F3A9157
http://purl.uniprot.org/uniprot/#_A0A0B6XK11-mappedCitation-25964490
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/CFAD8300F2868A5EC8CD44D1CE89A990963AEC6ADE3F6FE992123DB8F9D9A0674DD6E9872A3ABD0E299F9B982F3A9157