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DESCRIBE <http://purl.uniprot.org/SHA-384/CFE19099EDF9193A9D97EC2EF8C7F0838757790B3AE3D4980ED41CB27B966D1B0F6463FFB26C9CE324044925C298EE24>
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http://purl.uniprot.org/SHA-384/CFE19099EDF9193A9D97EC2EF8C7F0838757790B3AE3D4980ED41CB27B966D1B0F6463FFB26C9CE324044925C298EE24
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/CFE19099EDF9193A9D97EC2EF8C7F0838757790B3AE3D4980ED41CB27B966D1B0F6463FFB26C9CE324044925C298EE24
http://www.w3.org/2000/01/rdf-schema#comment
"our results demonstrate the central role played by GlyT2 in determining inhibitory phenotype and therefore in the physiology and pathology of inhibitory circuits."
xsd:string
http://purl.uniprot.org/uniprot/#_B3DB935EC4ECFC1D304F69BCA9EF93CCB9D26541B85B380F087D473B99627181D44C715C413D3FA00CB33C2EA777E0E0
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/CFE19099EDF9193A9D97EC2EF8C7F0838757790B3AE3D4980ED41CB27B966D1B0F6463FFB26C9CE324044925C298EE24
http://purl.uniprot.org/uniprot/B2RQX9
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/CFE19099EDF9193A9D97EC2EF8C7F0838757790B3AE3D4980ED41CB27B966D1B0F6463FFB26C9CE324044925C298EE24
http://purl.uniprot.org/uniprot/#_B2RQX9-mappedCitation-18815261
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/CFE19099EDF9193A9D97EC2EF8C7F0838757790B3AE3D4980ED41CB27B966D1B0F6463FFB26C9CE324044925C298EE24