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DESCRIBE <http://purl.uniprot.org/SHA-384/D0486E1A152A3F26C22C5E7DE696C3E092F86CB6FC6332F25C23EF87CD103E13738CA57F7229827F6727AD90B5248219>
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http://purl.uniprot.org/SHA-384/D0486E1A152A3F26C22C5E7DE696C3E092F86CB6FC6332F25C23EF87CD103E13738CA57F7229827F6727AD90B5248219
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/D0486E1A152A3F26C22C5E7DE696C3E092F86CB6FC6332F25C23EF87CD103E13738CA57F7229827F6727AD90B5248219
http://www.w3.org/2000/01/rdf-schema#comment
"mutations specifically disrupting hERG 1b function are expected to reduce cardiac I Kr and enhance drug sensitivity and represent a potential mechanism underlying inherited or acquired LQTS."
xsd:string
http://purl.uniprot.org/uniprot/#_318C76D235C9DCD7249FCF08FBD09F81B041ABD94F1252A7698E7DDB214C160A74CB171DACF35BC6760FADE79F7AB931
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/D0486E1A152A3F26C22C5E7DE696C3E092F86CB6FC6332F25C23EF87CD103E13738CA57F7229827F6727AD90B5248219
http://purl.uniprot.org/uniprot/Q12809
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/D0486E1A152A3F26C22C5E7DE696C3E092F86CB6FC6332F25C23EF87CD103E13738CA57F7229827F6727AD90B5248219
http://purl.uniprot.org/uniprot/#_Q12809-mappedCitation-18776039
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/D0486E1A152A3F26C22C5E7DE696C3E092F86CB6FC6332F25C23EF87CD103E13738CA57F7229827F6727AD90B5248219