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DESCRIBE <http://purl.uniprot.org/SHA-384/D128CA3B702B2A4648BC838DDFE8EEBB41A7125DF171154D858363EE11C7A4E660A419A016B334407DD9308F28F613B3>
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http://purl.uniprot.org/SHA-384/D128CA3B702B2A4648BC838DDFE8EEBB41A7125DF171154D858363EE11C7A4E660A419A016B334407DD9308F28F613B3
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/D128CA3B702B2A4648BC838DDFE8EEBB41A7125DF171154D858363EE11C7A4E660A419A016B334407DD9308F28F613B3
http://www.w3.org/2000/01/rdf-schema#comment
"Loss of Hox11 function leads to fracture repair defects including reduced cartilage formation and delayed ossification."
xsd:string
http://purl.uniprot.org/uniprot/#_FA6D4C79E78C5C9858223D4A33FB9AD6C77270087E4C13ECFC9AC1A3927AC7230294818042542821EF7F8E747E52F005
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/D128CA3B702B2A4648BC838DDFE8EEBB41A7125DF171154D858363EE11C7A4E660A419A016B334407DD9308F28F613B3
http://purl.uniprot.org/uniprot/Q9QX99
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/D128CA3B702B2A4648BC838DDFE8EEBB41A7125DF171154D858363EE11C7A4E660A419A016B334407DD9308F28F613B3
http://purl.uniprot.org/uniprot/#_Q9QX99-mappedCitation-27939685
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/D128CA3B702B2A4648BC838DDFE8EEBB41A7125DF171154D858363EE11C7A4E660A419A016B334407DD9308F28F613B3