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DESCRIBE <http://purl.uniprot.org/SHA-384/D2733C47D0694BCCE1B63B648E27FFA7A8BE6EBF0B6B55223B38529CE8E0C8C7334F1EE1DB853EC4D6A734F467D63337>
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http://purl.uniprot.org/SHA-384/D2733C47D0694BCCE1B63B648E27FFA7A8BE6EBF0B6B55223B38529CE8E0C8C7334F1EE1DB853EC4D6A734F467D63337
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/D2733C47D0694BCCE1B63B648E27FFA7A8BE6EBF0B6B55223B38529CE8E0C8C7334F1EE1DB853EC4D6A734F467D63337
http://www.w3.org/2000/01/rdf-schema#comment
"FLCN deficiency and subsequent increased PPARGC1A expression result in increased mitochondrial function and oxidative metabolism as the source of cellular energy which may drive hyperplastic transformation."
xsd:string
http://purl.uniprot.org/uniprot/#_0B3114DDF5C20585212D71FEC7623464E74E45BDCA934525AD42B826ED53380BB60DF232554FE3354FAD03C1A4B431C1
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/D2733C47D0694BCCE1B63B648E27FFA7A8BE6EBF0B6B55223B38529CE8E0C8C7334F1EE1DB853EC4D6A734F467D63337
http://purl.uniprot.org/uniprot/Q8NFG4
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/D2733C47D0694BCCE1B63B648E27FFA7A8BE6EBF0B6B55223B38529CE8E0C8C7334F1EE1DB853EC4D6A734F467D63337
http://purl.uniprot.org/uniprot/#_Q8NFG4-mappedCitation-23150719
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/D2733C47D0694BCCE1B63B648E27FFA7A8BE6EBF0B6B55223B38529CE8E0C8C7334F1EE1DB853EC4D6A734F467D63337