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http://purl.uniprot.org/SHA-384/D2A81662449DCD989CE6D9AE6D1DB2E64BF5FD27A9E1A443BCD10A1962162D07E75DEE255A4308CF8CCC1F9D5968BD31http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/D2A81662449DCD989CE6D9AE6D1DB2E64BF5FD27A9E1A443BCD10A1962162D07E75DEE255A4308CF8CCC1F9D5968BD31http://www.w3.org/2000/01/rdf-schema#comment"Variant:p.Cys2664Thr. Disease mentioned:Alzheimer's disease[DOID:10652]. Impact:Risk to Disease. Evidence sentence:The distribution of the NR2b genotypes (p = 0.600) and alleles (p = 0.652) did not differ significantly between AD patients and controls, however, suggesting that it is unlikely that the NR2b C2664T polymorphism plays a substantial role in conferring susceptibility to AD. Abstract Section: RESULTS."xsd:string
http://purl.uniprot.org/uniprot/#_2AE72651AA7AC9B656E73E692551AECCA865ACF6AF30BF38EE62C47E37FA3404FB9D6129CBAA32F4326450CDCD2C01C4http://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/D2A81662449DCD989CE6D9AE6D1DB2E64BF5FD27A9E1A443BCD10A1962162D07E75DEE255A4308CF8CCC1F9D5968BD31
http://purl.uniprot.org/uniprot/Q13224http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/D2A81662449DCD989CE6D9AE6D1DB2E64BF5FD27A9E1A443BCD10A1962162D07E75DEE255A4308CF8CCC1F9D5968BD31
http://purl.uniprot.org/uniprot/#_Q13224-mappedCitation-11844890http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/D2A81662449DCD989CE6D9AE6D1DB2E64BF5FD27A9E1A443BCD10A1962162D07E75DEE255A4308CF8CCC1F9D5968BD31