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DESCRIBE <http://purl.uniprot.org/SHA-384/D2BAB355A189B4D45C439BFCAB30F3ED1AFAE5D7251417DFD09A0E608E8B65696DEBCFDA0EF33926C948EA54039D77E3>
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http://purl.uniprot.org/SHA-384/D2BAB355A189B4D45C439BFCAB30F3ED1AFAE5D7251417DFD09A0E608E8B65696DEBCFDA0EF33926C948EA54039D77E3
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/D2BAB355A189B4D45C439BFCAB30F3ED1AFAE5D7251417DFD09A0E608E8B65696DEBCFDA0EF33926C948EA54039D77E3
http://www.w3.org/2000/01/rdf-schema#comment
"These findings demonstrate that the genes involved in dictating thiamine homeostasis such as SLC19A2 SLC25A19 and TPK-1 were significantly up-regulated in clinical tissues and breast cancer cell lines."
xsd:string
http://purl.uniprot.org/uniprot/#_ED5545921E77044C52910947BB8F76D130788FBEE21EB60965FBAA5D102737E74CBA35F75DBABF08859EFD8FE7E066E8
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/D2BAB355A189B4D45C439BFCAB30F3ED1AFAE5D7251417DFD09A0E608E8B65696DEBCFDA0EF33926C948EA54039D77E3
http://purl.uniprot.org/uniprot/A0A2R8Y5B5
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/D2BAB355A189B4D45C439BFCAB30F3ED1AFAE5D7251417DFD09A0E608E8B65696DEBCFDA0EF33926C948EA54039D77E3
http://purl.uniprot.org/uniprot/#_A0A2R8Y5B5-mappedCitation-23642734
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/D2BAB355A189B4D45C439BFCAB30F3ED1AFAE5D7251417DFD09A0E608E8B65696DEBCFDA0EF33926C948EA54039D77E3