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DESCRIBE <http://purl.uniprot.org/SHA-384/D653BBE0E2420C347BAB55A2CB409B926C903A871683D68E55914E888EB2DC08625552A4800429EEA59BE569C7938309>
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http://purl.uniprot.org/SHA-384/D653BBE0E2420C347BAB55A2CB409B926C903A871683D68E55914E888EB2DC08625552A4800429EEA59BE569C7938309
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/D653BBE0E2420C347BAB55A2CB409B926C903A871683D68E55914E888EB2DC08625552A4800429EEA59BE569C7938309
http://www.w3.org/2000/01/rdf-schema#comment
"findings suggest ECM1 is released from infiltrating inflammatory cells which leads to cardiac fibroblast stimulation and fibrosis in aging and myocardial infarction. ECM1 may be a novel intermediary between inflammation and fibrosis."
xsd:string
http://purl.uniprot.org/uniprot/#_29ED40B48A986817BA5FB088A60A58BFA8A706E36EAA8CEB39A0DE943A9D986067B656CA3790ACD1EFBA698B04B88D28
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/D653BBE0E2420C347BAB55A2CB409B926C903A871683D68E55914E888EB2DC08625552A4800429EEA59BE569C7938309
http://purl.uniprot.org/uniprot/Q61508
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/D653BBE0E2420C347BAB55A2CB409B926C903A871683D68E55914E888EB2DC08625552A4800429EEA59BE569C7938309
http://purl.uniprot.org/uniprot/#_Q61508-mappedCitation-30789914
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/D653BBE0E2420C347BAB55A2CB409B926C903A871683D68E55914E888EB2DC08625552A4800429EEA59BE569C7938309