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DESCRIBE <http://purl.uniprot.org/SHA-384/D6A5D12AD0B7707251E47D5619B577E6BB28C7FFC1218C3A9444C596AAC250BD06DF0526654D047FD6D375A999AECBB7>
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http://purl.uniprot.org/SHA-384/D6A5D12AD0B7707251E47D5619B577E6BB28C7FFC1218C3A9444C596AAC250BD06DF0526654D047FD6D375A999AECBB7
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/D6A5D12AD0B7707251E47D5619B577E6BB28C7FFC1218C3A9444C596AAC250BD06DF0526654D047FD6D375A999AECBB7
http://www.w3.org/2000/01/rdf-schema#comment
"the results indicated that miR145 serves a critical role in suppressing the biological behavior of EOC cells by targeting CCND2 and E2F3."
xsd:string
http://purl.uniprot.org/uniprot/#_A9F6C3990FAD116BA16BDA983A011CDFC15E285A8CC5A5AAA7808CD45AFFA8F33588E3F445ED6C9ED8EB787495A68BF4
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/D6A5D12AD0B7707251E47D5619B577E6BB28C7FFC1218C3A9444C596AAC250BD06DF0526654D047FD6D375A999AECBB7
http://purl.uniprot.org/uniprot/Q24JQ3
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/D6A5D12AD0B7707251E47D5619B577E6BB28C7FFC1218C3A9444C596AAC250BD06DF0526654D047FD6D375A999AECBB7
http://purl.uniprot.org/uniprot/#_Q24JQ3-mappedCitation-30864742
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/D6A5D12AD0B7707251E47D5619B577E6BB28C7FFC1218C3A9444C596AAC250BD06DF0526654D047FD6D375A999AECBB7