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DESCRIBE <http://purl.uniprot.org/SHA-384/D6E87F88D4626C42C20B1A5FAD15E43FD87C7B03E86C2240F952D6D8D19B58E27C18A206424AD1F7F5FD23BBAB265FC7>
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http://purl.uniprot.org/SHA-384/D6E87F88D4626C42C20B1A5FAD15E43FD87C7B03E86C2240F952D6D8D19B58E27C18A206424AD1F7F5FD23BBAB265FC7
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/D6E87F88D4626C42C20B1A5FAD15E43FD87C7B03E86C2240F952D6D8D19B58E27C18A206424AD1F7F5FD23BBAB265FC7
http://www.w3.org/2000/01/rdf-schema#comment
"IL-17A and IL-17F secreted by CD4+Th17 cells specific to lung self-antigens are critical mediators of autoimmunity leading to the pathogenesis of obliterative airway disease."
xsd:string
http://purl.uniprot.org/uniprot/#_5565C05A3417EA345D3CF013882B41C62DE17D50033590E1934C9E3EB136A36096AD698AF48CB6435C8E71D12527F7FF
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/D6E87F88D4626C42C20B1A5FAD15E43FD87C7B03E86C2240F952D6D8D19B58E27C18A206424AD1F7F5FD23BBAB265FC7
http://purl.uniprot.org/uniprot/Q7TNI7
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/D6E87F88D4626C42C20B1A5FAD15E43FD87C7B03E86C2240F952D6D8D19B58E27C18A206424AD1F7F5FD23BBAB265FC7
http://purl.uniprot.org/uniprot/#_Q7TNI7-mappedCitation-23325004
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/D6E87F88D4626C42C20B1A5FAD15E43FD87C7B03E86C2240F952D6D8D19B58E27C18A206424AD1F7F5FD23BBAB265FC7