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DESCRIBE <http://purl.uniprot.org/SHA-384/D962021F18E6BD6C4A9889673513A12E9CBB6DDF594274EF77AC02E3F7FEBB3B2E7AC7AA065FF1523935BADDAA31B5B1>
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http://purl.uniprot.org/SHA-384/D962021F18E6BD6C4A9889673513A12E9CBB6DDF594274EF77AC02E3F7FEBB3B2E7AC7AA065FF1523935BADDAA31B5B1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/D962021F18E6BD6C4A9889673513A12E9CBB6DDF594274EF77AC02E3F7FEBB3B2E7AC7AA065FF1523935BADDAA31B5B1
http://www.w3.org/2000/01/rdf-schema#comment
"The DES-p.Glu401Asp mutation causes predominant inherited left ventricular arrhythmogenic cardiomyopathy/dysplasia with a high incidence of adverse clinical events in the absence of skeletal myopathy or conduction system disorders."
xsd:string
http://purl.uniprot.org/uniprot/#_DD666A0D8B2E06BED4B6CAA53E68BD3E5051129340C984C049E591CF9D444AB32F3C1313062B633772584839F1A1B7EF
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/D962021F18E6BD6C4A9889673513A12E9CBB6DDF594274EF77AC02E3F7FEBB3B2E7AC7AA065FF1523935BADDAA31B5B1
http://purl.uniprot.org/uniprot/A5Z217
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/D962021F18E6BD6C4A9889673513A12E9CBB6DDF594274EF77AC02E3F7FEBB3B2E7AC7AA065FF1523935BADDAA31B5B1
http://purl.uniprot.org/uniprot/#_A5Z217-mappedCitation-29212896
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/D962021F18E6BD6C4A9889673513A12E9CBB6DDF594274EF77AC02E3F7FEBB3B2E7AC7AA065FF1523935BADDAA31B5B1