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DESCRIBE <http://purl.uniprot.org/SHA-384/DA89C5536534527047AA1DB4E665573891A261F3BBDFF90BB9893CBCDF04BDA4272B9EDDD645F430763240D890CD7567>
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http://purl.uniprot.org/SHA-384/DA89C5536534527047AA1DB4E665573891A261F3BBDFF90BB9893CBCDF04BDA4272B9EDDD645F430763240D890CD7567
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/DA89C5536534527047AA1DB4E665573891A261F3BBDFF90BB9893CBCDF04BDA4272B9EDDD645F430763240D890CD7567
http://www.w3.org/2000/01/rdf-schema#comment
"These findings indicate that K(ATP) channels in alpha-cells play a key role in regulating glucagon secretion thereby adding to the paradox of how mice that lack K(ATP) channels maintain euglycemia."
xsd:string
http://purl.uniprot.org/uniprot/#_FBDB7BBF6E082C637680012B2E61BEB7D9090DD39C54155B243A9FA526AD7F06F4BD4EB0FB35E3D00FFE7826E2308BE9
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/DA89C5536534527047AA1DB4E665573891A261F3BBDFF90BB9893CBCDF04BDA4272B9EDDD645F430763240D890CD7567
http://purl.uniprot.org/uniprot/B2RUS7
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/DA89C5536534527047AA1DB4E665573891A261F3BBDFF90BB9893CBCDF04BDA4272B9EDDD645F430763240D890CD7567
http://purl.uniprot.org/uniprot/#_B2RUS7-mappedCitation-15941784
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/DA89C5536534527047AA1DB4E665573891A261F3BBDFF90BB9893CBCDF04BDA4272B9EDDD645F430763240D890CD7567