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DESCRIBE <http://purl.uniprot.org/SHA-384/DC29E255ADE34512136CDB7CF3C1D0E0F4BE3E80D47058F4C1E26C682A2C42E4D8D3C283F5D3B495DAE3ADCFF170C9FD>
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http://purl.uniprot.org/SHA-384/DC29E255ADE34512136CDB7CF3C1D0E0F4BE3E80D47058F4C1E26C682A2C42E4D8D3C283F5D3B495DAE3ADCFF170C9FD
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/DC29E255ADE34512136CDB7CF3C1D0E0F4BE3E80D47058F4C1E26C682A2C42E4D8D3C283F5D3B495DAE3ADCFF170C9FD
http://www.w3.org/2000/01/rdf-schema#comment
"both 1alphaOHase gene ablation and Pi supplementation inhibit renal calcification in Npt2-/- mice and that 1 25(OH)2D is essential for the development of hypercalciuria and nephrocalcinosis in the mutant strain."
xsd:string
http://purl.uniprot.org/uniprot/#_354D84CFB5A83DA6BB6B8D98E2C5701166C1A599E4E1F9C5C21ACB4C7B80B849A4749DEF997B7DC8D17EBF9B63FFA940
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/DC29E255ADE34512136CDB7CF3C1D0E0F4BE3E80D47058F4C1E26C682A2C42E4D8D3C283F5D3B495DAE3ADCFF170C9FD
http://purl.uniprot.org/uniprot/Q9D2V6
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/DC29E255ADE34512136CDB7CF3C1D0E0F4BE3E80D47058F4C1E26C682A2C42E4D8D3C283F5D3B495DAE3ADCFF170C9FD
http://purl.uniprot.org/uniprot/#_Q9D2V6-mappedCitation-14656762
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/DC29E255ADE34512136CDB7CF3C1D0E0F4BE3E80D47058F4C1E26C682A2C42E4D8D3C283F5D3B495DAE3ADCFF170C9FD