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http://purl.uniprot.org/SHA-384/DF16F0DEEE622533087FA3B8E059E90573C949BAFFEA94A883021523E2088D4738CB348AFFD5B5ECF3D36C1B5B4F10B1http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/DF16F0DEEE622533087FA3B8E059E90573C949BAFFEA94A883021523E2088D4738CB348AFFD5B5ECF3D36C1B5B4F10B1http://www.w3.org/2000/01/rdf-schema#comment"Study reports that hyperinsulinemia could induce persistent Egr-1 activation in hepatocytes which provides negative feedback inhibition of insulin sensitivity by inducing the expression of PTP1B. Mechanistically Egr-1 inhibits insulin receptor phosphorylation by directly activating PTP1B transcription in the liver."xsd:string
http://purl.uniprot.org/uniprot/#_75B758C73200F97B412188FE17C57D8C1B02DB880217AA0FFBA0426E56FC210D921A8269C8755FC983CC4F8D95E2A6F6http://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/DF16F0DEEE622533087FA3B8E059E90573C949BAFFEA94A883021523E2088D4738CB348AFFD5B5ECF3D36C1B5B4F10B1
http://purl.uniprot.org/uniprot/Q3TZW9http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/DF16F0DEEE622533087FA3B8E059E90573C949BAFFEA94A883021523E2088D4738CB348AFFD5B5ECF3D36C1B5B4F10B1
http://purl.uniprot.org/uniprot/#_Q3TZW9-mappedCitation-31309546http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/DF16F0DEEE622533087FA3B8E059E90573C949BAFFEA94A883021523E2088D4738CB348AFFD5B5ECF3D36C1B5B4F10B1