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DESCRIBE <http://purl.uniprot.org/SHA-384/E073E79DFF92D4F7A2AC319A0EDD5D3E775178699A4EA11148F1071E5A6A79DAB69DC4B24CAA21272B2AB44F6B1CE1FA>
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http://purl.uniprot.org/SHA-384/E073E79DFF92D4F7A2AC319A0EDD5D3E775178699A4EA11148F1071E5A6A79DAB69DC4B24CAA21272B2AB44F6B1CE1FA
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/E073E79DFF92D4F7A2AC319A0EDD5D3E775178699A4EA11148F1071E5A6A79DAB69DC4B24CAA21272B2AB44F6B1CE1FA
http://www.w3.org/2000/01/rdf-schema#comment
"Mechanical wounding induces significant ROS generation at the wound edge which in turn induced ligand-independent KGFR and FRS2 activation via c-Src kinase signaling."
xsd:string
http://purl.uniprot.org/uniprot/#_CC22F42FA82309D2E3074288DB3897D7AA4F904761B17B3EDE88B5117E72966FEEF0E94F8C700C644AA80CBCC26355C3
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/E073E79DFF92D4F7A2AC319A0EDD5D3E775178699A4EA11148F1071E5A6A79DAB69DC4B24CAA21272B2AB44F6B1CE1FA
http://purl.uniprot.org/uniprot/L7RTG7
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/E073E79DFF92D4F7A2AC319A0EDD5D3E775178699A4EA11148F1071E5A6A79DAB69DC4B24CAA21272B2AB44F6B1CE1FA
http://purl.uniprot.org/uniprot/#_L7RTG7-mappedCitation-19111446
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/E073E79DFF92D4F7A2AC319A0EDD5D3E775178699A4EA11148F1071E5A6A79DAB69DC4B24CAA21272B2AB44F6B1CE1FA