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DESCRIBE <http://purl.uniprot.org/SHA-384/E0DF22CA6396E65150853F95A38F16AD2CD282E4AEFE0D95DFD3447C242C3B44F65412570C6D4E752C39091B2129421E>
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http://purl.uniprot.org/SHA-384/E0DF22CA6396E65150853F95A38F16AD2CD282E4AEFE0D95DFD3447C242C3B44F65412570C6D4E752C39091B2129421E
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/E0DF22CA6396E65150853F95A38F16AD2CD282E4AEFE0D95DFD3447C242C3B44F65412570C6D4E752C39091B2129421E
http://www.w3.org/2000/01/rdf-schema#comment
"loss of BBS1 BBS4 or OFD1 led to decreased NF-kappaB activity and concomitant IkappaBbeta accumulation and that these defects were ameliorated with SFN treatment."
xsd:string
http://purl.uniprot.org/uniprot/#_DC3B3CC377B9BFAD49E6B85584B24A4E57709144D2E5AAE2CB36091642AD5C62BA6C32230DB3B980A87F0A07175BC71F
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/E0DF22CA6396E65150853F95A38F16AD2CD282E4AEFE0D95DFD3447C242C3B44F65412570C6D4E752C39091B2129421E
http://purl.uniprot.org/uniprot/A8K2T9
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/E0DF22CA6396E65150853F95A38F16AD2CD282E4AEFE0D95DFD3447C242C3B44F65412570C6D4E752C39091B2129421E
http://purl.uniprot.org/uniprot/#_A8K2T9-mappedCitation-24691443
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/E0DF22CA6396E65150853F95A38F16AD2CD282E4AEFE0D95DFD3447C242C3B44F65412570C6D4E752C39091B2129421E