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DESCRIBE <http://purl.uniprot.org/SHA-384/E4FA9274F6265A97B66AB7B8F401E1B5C0D6E55AF666134667E445A21882195C98A9B671D33E83D157F014BA6980DEB1>
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http://purl.uniprot.org/SHA-384/E4FA9274F6265A97B66AB7B8F401E1B5C0D6E55AF666134667E445A21882195C98A9B671D33E83D157F014BA6980DEB1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/E4FA9274F6265A97B66AB7B8F401E1B5C0D6E55AF666134667E445A21882195C98A9B671D33E83D157F014BA6980DEB1
http://www.w3.org/2000/01/rdf-schema#comment
"These results support the potential therapeutic use of FGF2 or related molecules in the treatment of MDD and point to alternate mechanisms of neuronal remodeling that may be critical in this treatment."
xsd:string
http://purl.uniprot.org/uniprot/#_9CB14F4BA7DD8F55C92127F35CA6F1AB4A170D6F80C46031C713F8F08C1C6C423B55E6E05CAFEE38B37CCA190B3AA2B2
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/E4FA9274F6265A97B66AB7B8F401E1B5C0D6E55AF666134667E445A21882195C98A9B671D33E83D157F014BA6980DEB1
http://purl.uniprot.org/uniprot/P13109
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/E4FA9274F6265A97B66AB7B8F401E1B5C0D6E55AF666134667E445A21882195C98A9B671D33E83D157F014BA6980DEB1
http://purl.uniprot.org/uniprot/#_P13109-mappedCitation-18586016
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/E4FA9274F6265A97B66AB7B8F401E1B5C0D6E55AF666134667E445A21882195C98A9B671D33E83D157F014BA6980DEB1