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DESCRIBE <http://purl.uniprot.org/SHA-384/E518E25E2CCB55E20306B1B9F4E191C203E88B7FC7C613C3F04AE264559BDF04AD09701DF09841059561881E81DBAE43>
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http://purl.uniprot.org/SHA-384/E518E25E2CCB55E20306B1B9F4E191C203E88B7FC7C613C3F04AE264559BDF04AD09701DF09841059561881E81DBAE43
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/E518E25E2CCB55E20306B1B9F4E191C203E88B7FC7C613C3F04AE264559BDF04AD09701DF09841059561881E81DBAE43
http://www.w3.org/2000/01/rdf-schema#comment
"Increased CF6 may be responsible in part for decreased prostacyclin observed in coronary heart disease in particular after PTCA and stent therapy. Potential risk factor for coronary heart disease."
xsd:string
http://purl.uniprot.org/uniprot/#_E8A4E618DBF7788A9B46CFF06102F48F81D7D51A4B55F027F65A643C864A504BD1F5A0312EB218A0987B8A053A9EDCD8
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/E518E25E2CCB55E20306B1B9F4E191C203E88B7FC7C613C3F04AE264559BDF04AD09701DF09841059561881E81DBAE43
http://purl.uniprot.org/uniprot/Q6NZ59
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/E518E25E2CCB55E20306B1B9F4E191C203E88B7FC7C613C3F04AE264559BDF04AD09701DF09841059561881E81DBAE43
http://purl.uniprot.org/uniprot/#_Q6NZ59-mappedCitation-17456993
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/E518E25E2CCB55E20306B1B9F4E191C203E88B7FC7C613C3F04AE264559BDF04AD09701DF09841059561881E81DBAE43