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DESCRIBE <http://purl.uniprot.org/SHA-384/E76A117A4CF70DB58A075AB006C911BE14FFF66A5D367D7A6E4CC0DAE1F47A7C5937E54CE7DF3A4833774DD25FBC2DA5>
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http://purl.uniprot.org/SHA-384/E76A117A4CF70DB58A075AB006C911BE14FFF66A5D367D7A6E4CC0DAE1F47A7C5937E54CE7DF3A4833774DD25FBC2DA5
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/E76A117A4CF70DB58A075AB006C911BE14FFF66A5D367D7A6E4CC0DAE1F47A7C5937E54CE7DF3A4833774DD25FBC2DA5
http://www.w3.org/2000/01/rdf-schema#comment
"results demonstrate for the first time that N-Myc is a downstream target of RET2A signaling and propose that induction of N-Myc by RET2A is a key step leading to lower p18 levels during MEN2A tumorigenesis"
xsd:string
http://purl.uniprot.org/uniprot/#_D85588072074F89EAF39EAAF5482C8E4BC3FC8E7AC3F23FBB129A282564432C2F5BE0A19CB2D9956E80B4C95665B4572
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/E76A117A4CF70DB58A075AB006C911BE14FFF66A5D367D7A6E4CC0DAE1F47A7C5937E54CE7DF3A4833774DD25FBC2DA5
http://purl.uniprot.org/uniprot/M9Z0T2
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/E76A117A4CF70DB58A075AB006C911BE14FFF66A5D367D7A6E4CC0DAE1F47A7C5937E54CE7DF3A4833774DD25FBC2DA5
http://purl.uniprot.org/uniprot/#_M9Z0T2-mappedCitation-21112821
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/E76A117A4CF70DB58A075AB006C911BE14FFF66A5D367D7A6E4CC0DAE1F47A7C5937E54CE7DF3A4833774DD25FBC2DA5