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DESCRIBE <http://purl.uniprot.org/SHA-384/E8C3B6D1F0A4EE90DA192587F76F46FEFC2F9D5DDDB70DE3454B1130C337E0B887C7C34FA1819F4F6320C1CE6E7B2E6E>
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http://purl.uniprot.org/SHA-384/E8C3B6D1F0A4EE90DA192587F76F46FEFC2F9D5DDDB70DE3454B1130C337E0B887C7C34FA1819F4F6320C1CE6E7B2E6E
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/E8C3B6D1F0A4EE90DA192587F76F46FEFC2F9D5DDDB70DE3454B1130C337E0B887C7C34FA1819F4F6320C1CE6E7B2E6E
http://www.w3.org/2000/01/rdf-schema#comment
"These results suggested that Foxo1 overexpression in betacells was able to increase apoptosis by inhibiting CD24 expression. This study may provide an approach for the treatment and prevention of type 2 diabetes."
xsd:string
http://purl.uniprot.org/uniprot/#_3C7FDBBC53492D1FBBE1C55B7CC7D9541F493A19CCE45405F5279403C3579B01798EFFCF5104FBB3D0F1FE1D83FD0F5C
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/E8C3B6D1F0A4EE90DA192587F76F46FEFC2F9D5DDDB70DE3454B1130C337E0B887C7C34FA1819F4F6320C1CE6E7B2E6E
http://purl.uniprot.org/uniprot/Q07490
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/E8C3B6D1F0A4EE90DA192587F76F46FEFC2F9D5DDDB70DE3454B1130C337E0B887C7C34FA1819F4F6320C1CE6E7B2E6E
http://purl.uniprot.org/uniprot/#_Q07490-mappedCitation-26935354
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/E8C3B6D1F0A4EE90DA192587F76F46FEFC2F9D5DDDB70DE3454B1130C337E0B887C7C34FA1819F4F6320C1CE6E7B2E6E