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DESCRIBE <http://purl.uniprot.org/SHA-384/E98682D18C30624248963E3FB214E03CB643D3D10CFCB7315911B5E995EAA7F510945C3A6B48E13E8C37920D67A2FA42>
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http://purl.uniprot.org/SHA-384/E98682D18C30624248963E3FB214E03CB643D3D10CFCB7315911B5E995EAA7F510945C3A6B48E13E8C37920D67A2FA42
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/E98682D18C30624248963E3FB214E03CB643D3D10CFCB7315911B5E995EAA7F510945C3A6B48E13E8C37920D67A2FA42
http://www.w3.org/2000/01/rdf-schema#comment
"Knockdown of endogenous Shank2E or overexpression of a dominant-negative Shank2E mutant inhibited the glucocorticoid-mediated increase in CFTR."
xsd:string
http://purl.uniprot.org/uniprot/#_8C0674CC34E99DFCC426AE3176F1E1AC1932A0B783F863530172DFB92BDD4AF1BEBAE50EE24759A274BCF58C4BE744E7
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/E98682D18C30624248963E3FB214E03CB643D3D10CFCB7315911B5E995EAA7F510945C3A6B48E13E8C37920D67A2FA42
http://purl.uniprot.org/uniprot/B7ZKU9
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/E98682D18C30624248963E3FB214E03CB643D3D10CFCB7315911B5E995EAA7F510945C3A6B48E13E8C37920D67A2FA42
http://purl.uniprot.org/uniprot/#_B7ZKU9-mappedCitation-24811177
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/E98682D18C30624248963E3FB214E03CB643D3D10CFCB7315911B5E995EAA7F510945C3A6B48E13E8C37920D67A2FA42