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DESCRIBE <http://purl.uniprot.org/SHA-384/EC8D2ACBE1B563A61EB75264B9E58229C1888E96E84958FD1DBA1B864660D1D35ABE1E2EFAEEAC221B9EF6802C493CA7>
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http://purl.uniprot.org/SHA-384/EC8D2ACBE1B563A61EB75264B9E58229C1888E96E84958FD1DBA1B864660D1D35ABE1E2EFAEEAC221B9EF6802C493CA7
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/EC8D2ACBE1B563A61EB75264B9E58229C1888E96E84958FD1DBA1B864660D1D35ABE1E2EFAEEAC221B9EF6802C493CA7
http://www.w3.org/2000/01/rdf-schema#comment
"we predict that the gene silencing of GSN and/or the downstream blocking of GSN along the p38 pathway could be applied to ameliorate pathological cardiac hypertrophy in the future"
xsd:string
http://purl.uniprot.org/uniprot/#_7F9E256E9B5CC81AFF9A3C3E5C64AE641D7AB8728E9BC85B1B51C01F6BDE430DE089550A60FDE709E226123F0A26DB0C
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/EC8D2ACBE1B563A61EB75264B9E58229C1888E96E84958FD1DBA1B864660D1D35ABE1E2EFAEEAC221B9EF6802C493CA7
http://purl.uniprot.org/uniprot/A0A384MEF1
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/EC8D2ACBE1B563A61EB75264B9E58229C1888E96E84958FD1DBA1B864660D1D35ABE1E2EFAEEAC221B9EF6802C493CA7
http://purl.uniprot.org/uniprot/#_A0A384MEF1-mappedCitation-24505034
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/EC8D2ACBE1B563A61EB75264B9E58229C1888E96E84958FD1DBA1B864660D1D35ABE1E2EFAEEAC221B9EF6802C493CA7