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DESCRIBE <http://purl.uniprot.org/SHA-384/ED3BCE9D399F4581010FA429E7D8A2BF18C8C9DA36E5A1BA45CF2B922B83A5A592B49FBC7BB38E679608AFC06007D2D0>
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http://purl.uniprot.org/SHA-384/ED3BCE9D399F4581010FA429E7D8A2BF18C8C9DA36E5A1BA45CF2B922B83A5A592B49FBC7BB38E679608AFC06007D2D0
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/ED3BCE9D399F4581010FA429E7D8A2BF18C8C9DA36E5A1BA45CF2B922B83A5A592B49FBC7BB38E679608AFC06007D2D0
http://www.w3.org/2000/01/rdf-schema#comment
"TXNIP induction plays a critical role in beta-cell apoptosis and sugessts that inhibition of TSNIP may represent a novel approach to reduce glucotoxic beta-cell loss."
xsd:string
http://purl.uniprot.org/uniprot/#_AD2D891DBEAEA8DE03C4076D1750DC864353E0A26E119B0D331F67641780E6B31A6CE2A8582F4C9BB0FF3BBD474FA940
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/ED3BCE9D399F4581010FA429E7D8A2BF18C8C9DA36E5A1BA45CF2B922B83A5A592B49FBC7BB38E679608AFC06007D2D0
http://purl.uniprot.org/uniprot/Q91X82
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/ED3BCE9D399F4581010FA429E7D8A2BF18C8C9DA36E5A1BA45CF2B922B83A5A592B49FBC7BB38E679608AFC06007D2D0
http://purl.uniprot.org/uniprot/#_Q91X82-mappedCitation-18171713
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/ED3BCE9D399F4581010FA429E7D8A2BF18C8C9DA36E5A1BA45CF2B922B83A5A592B49FBC7BB38E679608AFC06007D2D0