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DESCRIBE <http://purl.uniprot.org/SHA-384/EDEF0E5A924ECCBCD03518500726B5D105D2E6C4A3A25216D64EFC5A92F244BFDF1514BE2F4AD065F3BB400FE07C7BD4>
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http://purl.uniprot.org/SHA-384/EDEF0E5A924ECCBCD03518500726B5D105D2E6C4A3A25216D64EFC5A92F244BFDF1514BE2F4AD065F3BB400FE07C7BD4
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/EDEF0E5A924ECCBCD03518500726B5D105D2E6C4A3A25216D64EFC5A92F244BFDF1514BE2F4AD065F3BB400FE07C7BD4
http://www.w3.org/2000/01/rdf-schema#comment
"We aimed to investigate whether TSN protected H9c2 cardiomyocytes from AngII-induced activation of IGF-2R pathway (IGF-2 and IGF-2R )and hypertrophy by mediating through Estrogen receptors"
xsd:string
http://purl.uniprot.org/uniprot/#_64E8A91CCD7D7B7F214258A1F82356088ACE8CD2C012E1DA9F28EA75DA7DF0C4FEF56CDCF38025D9B7F07FBD27DB0018
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/EDEF0E5A924ECCBCD03518500726B5D105D2E6C4A3A25216D64EFC5A92F244BFDF1514BE2F4AD065F3BB400FE07C7BD4
http://purl.uniprot.org/uniprot/P01346
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/EDEF0E5A924ECCBCD03518500726B5D105D2E6C4A3A25216D64EFC5A92F244BFDF1514BE2F4AD065F3BB400FE07C7BD4
http://purl.uniprot.org/uniprot/#_P01346-mappedCitation-28812967
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/EDEF0E5A924ECCBCD03518500726B5D105D2E6C4A3A25216D64EFC5A92F244BFDF1514BE2F4AD065F3BB400FE07C7BD4