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DESCRIBE <http://purl.uniprot.org/SHA-384/EF965DE6D0842B2BC8A54FCB359C4AE9A30BA44AC444BE7C6F6E6566B70E300F661799AE0CAA7E59D5E676D74B66FCEA>
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http://purl.uniprot.org/SHA-384/EF965DE6D0842B2BC8A54FCB359C4AE9A30BA44AC444BE7C6F6E6566B70E300F661799AE0CAA7E59D5E676D74B66FCEA
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/EF965DE6D0842B2BC8A54FCB359C4AE9A30BA44AC444BE7C6F6E6566B70E300F661799AE0CAA7E59D5E676D74B66FCEA
http://www.w3.org/2000/01/rdf-schema#comment
"FGL2 plays an important role in the pathogenesis of autoimmune myocarditis that also involves the PD-1/PD-L1 pathway. Our findings may provide novel therapeutic targets for the treatment of immune-induced heart injury."
xsd:string
http://purl.uniprot.org/uniprot/#_FEAC94AD6430849329AB51B299E903E0B6F9AAF4AF992D7E465E81D540881BC789AFCD6BB0F1835252834D630B6483E9
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/EF965DE6D0842B2BC8A54FCB359C4AE9A30BA44AC444BE7C6F6E6566B70E300F661799AE0CAA7E59D5E676D74B66FCEA
http://purl.uniprot.org/uniprot/Q32Q89
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/EF965DE6D0842B2BC8A54FCB359C4AE9A30BA44AC444BE7C6F6E6566B70E300F661799AE0CAA7E59D5E676D74B66FCEA
http://purl.uniprot.org/uniprot/#_Q32Q89-mappedCitation-28892130
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/EF965DE6D0842B2BC8A54FCB359C4AE9A30BA44AC444BE7C6F6E6566B70E300F661799AE0CAA7E59D5E676D74B66FCEA