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DESCRIBE <http://purl.uniprot.org/SHA-384/F0F1A466B009BCB92F2BCDCADA8F0C0B06929F1D049AB1E72B6F72F512AD303A31D62E1B5E2B3FF1E8330CE2B2C1226D>
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http://purl.uniprot.org/SHA-384/F0F1A466B009BCB92F2BCDCADA8F0C0B06929F1D049AB1E72B6F72F512AD303A31D62E1B5E2B3FF1E8330CE2B2C1226D
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/F0F1A466B009BCB92F2BCDCADA8F0C0B06929F1D049AB1E72B6F72F512AD303A31D62E1B5E2B3FF1E8330CE2B2C1226D
http://www.w3.org/2000/01/rdf-schema#comment
"GluR2 internalization may participate in the maintenance of N-methyl-D-aspartate receptor/protein kinase C-dependent nociceptive hypersensitivity in persistent inflammatory pain."
xsd:string
http://purl.uniprot.org/uniprot/#_A9274392D14225994E2B6E78E69908C00C8BCC1CB226C76FC063DBA3C57C1C39418C4D2DE01A2CD6EEBC6A395F9B46B7
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/F0F1A466B009BCB92F2BCDCADA8F0C0B06929F1D049AB1E72B6F72F512AD303A31D62E1B5E2B3FF1E8330CE2B2C1226D
http://purl.uniprot.org/uniprot/Q4LG64
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F0F1A466B009BCB92F2BCDCADA8F0C0B06929F1D049AB1E72B6F72F512AD303A31D62E1B5E2B3FF1E8330CE2B2C1226D
http://purl.uniprot.org/uniprot/#_Q4LG64-mappedCitation-19279258
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F0F1A466B009BCB92F2BCDCADA8F0C0B06929F1D049AB1E72B6F72F512AD303A31D62E1B5E2B3FF1E8330CE2B2C1226D