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DESCRIBE <http://purl.uniprot.org/SHA-384/F1FA18351B4DEA8C833DCC9EED211E023BEB3FB682C96CB897A75D6FBDAF33D9BB1E8E2FC2167785A822E4B5B1348C48>
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http://purl.uniprot.org/SHA-384/F1FA18351B4DEA8C833DCC9EED211E023BEB3FB682C96CB897A75D6FBDAF33D9BB1E8E2FC2167785A822E4B5B1348C48
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/F1FA18351B4DEA8C833DCC9EED211E023BEB3FB682C96CB897A75D6FBDAF33D9BB1E8E2FC2167785A822E4B5B1348C48
http://www.w3.org/2000/01/rdf-schema#comment
"Data indicate that reduced cardiac contractile and pulmonary vasoconstrictor responses via the ETB receptor are attenuated and that responses mediated by the ETA receptor are more prominent in the context of heart failure."
xsd:string
http://purl.uniprot.org/uniprot/#_98DFC6B3EAF1AAB0FA965B225916C85BF8693C8FCCC059CD3A5F849B476C733D8861A90305124222AF9B38CAC283F44C
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/F1FA18351B4DEA8C833DCC9EED211E023BEB3FB682C96CB897A75D6FBDAF33D9BB1E8E2FC2167785A822E4B5B1348C48
http://purl.uniprot.org/uniprot/P56497
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F1FA18351B4DEA8C833DCC9EED211E023BEB3FB682C96CB897A75D6FBDAF33D9BB1E8E2FC2167785A822E4B5B1348C48
http://purl.uniprot.org/uniprot/#_P56497-mappedCitation-15838318
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F1FA18351B4DEA8C833DCC9EED211E023BEB3FB682C96CB897A75D6FBDAF33D9BB1E8E2FC2167785A822E4B5B1348C48