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DESCRIBE <http://purl.uniprot.org/SHA-384/F30CF225E0C724B8F68E93B70CD422AEBE7ADE68E05A5FDAF57AA8BE59DBD172271D704962E3E7080F43B81B274FFF1F>
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http://purl.uniprot.org/SHA-384/F30CF225E0C724B8F68E93B70CD422AEBE7ADE68E05A5FDAF57AA8BE59DBD172271D704962E3E7080F43B81B274FFF1F
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/F30CF225E0C724B8F68E93B70CD422AEBE7ADE68E05A5FDAF57AA8BE59DBD172271D704962E3E7080F43B81B274FFF1F
http://www.w3.org/2000/01/rdf-schema#comment
"causes increases in nuclear NF-kappaB activity and secretion of MCP-1 and GM-CSF; studies implicate oncogene-induced cytokine-signaling pathways in a new mechanism linking inflammation with cancer"
xsd:string
http://purl.uniprot.org/uniprot/#_057DD87C2C2ABA68EF3DB35CC8E1156C5D22999EE4BC59801FFC910E4ACE29AF9B00B73F2AB6618C5A4A5BD7F794DA91
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/F30CF225E0C724B8F68E93B70CD422AEBE7ADE68E05A5FDAF57AA8BE59DBD172271D704962E3E7080F43B81B274FFF1F
http://purl.uniprot.org/uniprot/Q96E88
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F30CF225E0C724B8F68E93B70CD422AEBE7ADE68E05A5FDAF57AA8BE59DBD172271D704962E3E7080F43B81B274FFF1F
http://purl.uniprot.org/uniprot/#_Q96E88-mappedCitation-12881713
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F30CF225E0C724B8F68E93B70CD422AEBE7ADE68E05A5FDAF57AA8BE59DBD172271D704962E3E7080F43B81B274FFF1F