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DESCRIBE <http://purl.uniprot.org/SHA-384/F416836BA8E7690AE652179421D39D5A86DAD56A1D03D8F0B604440FBBBDB5C9B10C4BF327FC2455D0873F5A22284A7A>
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http://purl.uniprot.org/SHA-384/F416836BA8E7690AE652179421D39D5A86DAD56A1D03D8F0B604440FBBBDB5C9B10C4BF327FC2455D0873F5A22284A7A
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/F416836BA8E7690AE652179421D39D5A86DAD56A1D03D8F0B604440FBBBDB5C9B10C4BF327FC2455D0873F5A22284A7A
http://www.w3.org/2000/01/rdf-schema#comment
"Data suggest that TCF7L2 and Wnt signaling control gut and brain proglucagon expression and glucose homeostasis; positive cross-talk between Wnt and glucagon-like peptide 1/cAMP signaling appears to regulate brain metabolism."
xsd:string
http://purl.uniprot.org/uniprot/#_8A3EFE32A38DA844762FC60873903290003C32C3A78EAC672989BC3D2B8E65FDE3C738825C1A9A08D942CC62678004A0
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/F416836BA8E7690AE652179421D39D5A86DAD56A1D03D8F0B604440FBBBDB5C9B10C4BF327FC2455D0873F5A22284A7A
http://purl.uniprot.org/uniprot/Q3V1G9
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F416836BA8E7690AE652179421D39D5A86DAD56A1D03D8F0B604440FBBBDB5C9B10C4BF327FC2455D0873F5A22284A7A
http://purl.uniprot.org/uniprot/#_Q3V1G9-mappedCitation-22966074
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F416836BA8E7690AE652179421D39D5A86DAD56A1D03D8F0B604440FBBBDB5C9B10C4BF327FC2455D0873F5A22284A7A