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DESCRIBE <http://purl.uniprot.org/SHA-384/F4A41178AF3061A67F0D305C2B2B4106F118E7A5CFEB7A77639D0A3F423A9160E549B18FEE51BB2A3158C39C2113A735>
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http://purl.uniprot.org/SHA-384/F4A41178AF3061A67F0D305C2B2B4106F118E7A5CFEB7A77639D0A3F423A9160E549B18FEE51BB2A3158C39C2113A735
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/F4A41178AF3061A67F0D305C2B2B4106F118E7A5CFEB7A77639D0A3F423A9160E549B18FEE51BB2A3158C39C2113A735
http://www.w3.org/2000/01/rdf-schema#comment
"in addition to reduced glycolytic flux uncoupling of mitochondrial oxidative phosphorylation contributes to impaired nutrient-stimulated insulin secretion with either mutations or loss of HNF-1alpha"
xsd:string
http://purl.uniprot.org/uniprot/#_B7AE9DBD6F8E26EE2687E76F36DD72B92217FB8EDE9E5331D5B3C4F70F4EA2D940B58E00D8031D69A6F1DCA4A93D7B55
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/F4A41178AF3061A67F0D305C2B2B4106F118E7A5CFEB7A77639D0A3F423A9160E549B18FEE51BB2A3158C39C2113A735
http://purl.uniprot.org/uniprot/P22361
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F4A41178AF3061A67F0D305C2B2B4106F118E7A5CFEB7A77639D0A3F423A9160E549B18FEE51BB2A3158C39C2113A735
http://purl.uniprot.org/uniprot/#_P22361-mappedCitation-19376774
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F4A41178AF3061A67F0D305C2B2B4106F118E7A5CFEB7A77639D0A3F423A9160E549B18FEE51BB2A3158C39C2113A735