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DESCRIBE <http://purl.uniprot.org/SHA-384/F52BCE2F106E7868843CCD6DF49480F422AA3F9A6D5FFE28DE0EC4E4C3692502D8433CBD9D6C52DDE223DE1F0F060E38>
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http://purl.uniprot.org/SHA-384/F52BCE2F106E7868843CCD6DF49480F422AA3F9A6D5FFE28DE0EC4E4C3692502D8433CBD9D6C52DDE223DE1F0F060E38
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/F52BCE2F106E7868843CCD6DF49480F422AA3F9A6D5FFE28DE0EC4E4C3692502D8433CBD9D6C52DDE223DE1F0F060E38
http://www.w3.org/2000/01/rdf-schema#comment
"Data suggest that the interplay between CCR1 upregulation and inactivation of CXCR4 signaling (the CCR1/CXCR4 axis) drives the egress of multiple myeloma PCs from the bone marrow leading to disease dissemination."
xsd:string
http://purl.uniprot.org/uniprot/#_E6CBE3B696FC38AE4617729BEB8ED2C43B807FDABFE49497AD2E51F76D3551F736059DF3FFF3B918109951529AA987ED
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/F52BCE2F106E7868843CCD6DF49480F422AA3F9A6D5FFE28DE0EC4E4C3692502D8433CBD9D6C52DDE223DE1F0F060E38
http://purl.uniprot.org/uniprot/Q5U003
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F52BCE2F106E7868843CCD6DF49480F422AA3F9A6D5FFE28DE0EC4E4C3692502D8433CBD9D6C52DDE223DE1F0F060E38
http://purl.uniprot.org/uniprot/#_Q5U003-mappedCitation-28855206
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F52BCE2F106E7868843CCD6DF49480F422AA3F9A6D5FFE28DE0EC4E4C3692502D8433CBD9D6C52DDE223DE1F0F060E38