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DESCRIBE <http://purl.uniprot.org/SHA-384/F62B20F6188A7E9688C7E91742FEBAA49C0C31AFBDFA40D315CDD72E9FD8CF1E29BC1E77B88496853E21A7833EB386A9>
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http://purl.uniprot.org/SHA-384/F62B20F6188A7E9688C7E91742FEBAA49C0C31AFBDFA40D315CDD72E9FD8CF1E29BC1E77B88496853E21A7833EB386A9
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/F62B20F6188A7E9688C7E91742FEBAA49C0C31AFBDFA40D315CDD72E9FD8CF1E29BC1E77B88496853E21A7833EB386A9
http://www.w3.org/2000/01/rdf-schema#comment
"Accordingly in salt-dependent hypertension EOLC probably increases vascular resistance and blood pressure by reducing alpha2 Na(+) pump activity and promoting Ca(2)(+) entry via NCX in myocytes."
xsd:string
http://purl.uniprot.org/uniprot/#_B7C460A1EDEAB5F1653257154381598286B05C0821836C8BA8C85567A95F794D0D7F675777B691B4929182E91C3E5582
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/F62B20F6188A7E9688C7E91742FEBAA49C0C31AFBDFA40D315CDD72E9FD8CF1E29BC1E77B88496853E21A7833EB386A9
http://purl.uniprot.org/uniprot/Q6PIE5
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F62B20F6188A7E9688C7E91742FEBAA49C0C31AFBDFA40D315CDD72E9FD8CF1E29BC1E77B88496853E21A7833EB386A9
http://purl.uniprot.org/uniprot/#_Q6PIE5-mappedCitation-16166162
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F62B20F6188A7E9688C7E91742FEBAA49C0C31AFBDFA40D315CDD72E9FD8CF1E29BC1E77B88496853E21A7833EB386A9