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DESCRIBE <http://purl.uniprot.org/SHA-384/F655E5A4BA9BB5EB60B1D53974BF7F82C6E49D66B93FABC14C6F92BF0048DD3CE4330D150680CF8269A5508C3234C89D>
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http://purl.uniprot.org/SHA-384/F655E5A4BA9BB5EB60B1D53974BF7F82C6E49D66B93FABC14C6F92BF0048DD3CE4330D150680CF8269A5508C3234C89D
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/F655E5A4BA9BB5EB60B1D53974BF7F82C6E49D66B93FABC14C6F92BF0048DD3CE4330D150680CF8269A5508C3234C89D
http://www.w3.org/2000/01/rdf-schema#comment
"GBM cells overexpressing TNFAIP3 and NKIRAS2 were refractory to miR-125b-induced apoptosis resistance as well as TMZ resistance indicating that both genes are relevant targets of miR-125b."
xsd:string
http://purl.uniprot.org/uniprot/#_8C6C57481A98B71BC75B0FB238F6EE946E8C6F405A7A9ACA9F8D00DD6A6DDCDDA5F25C0115911A1ABF300AB755B45CFC
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/F655E5A4BA9BB5EB60B1D53974BF7F82C6E49D66B93FABC14C6F92BF0048DD3CE4330D150680CF8269A5508C3234C89D
http://purl.uniprot.org/uniprot/Q9NYR9
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F655E5A4BA9BB5EB60B1D53974BF7F82C6E49D66B93FABC14C6F92BF0048DD3CE4330D150680CF8269A5508C3234C89D
http://purl.uniprot.org/uniprot/#_Q9NYR9-mappedCitation-24901050
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F655E5A4BA9BB5EB60B1D53974BF7F82C6E49D66B93FABC14C6F92BF0048DD3CE4330D150680CF8269A5508C3234C89D