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DESCRIBE <http://purl.uniprot.org/SHA-384/F70CF10F1405D4FCDC24D6AD6D37C3CE7030DD3CAADF89460A76F50F4DEB18D6F445A80113DF050B0A1D3C68C957AEBC>
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http://purl.uniprot.org/SHA-384/F70CF10F1405D4FCDC24D6AD6D37C3CE7030DD3CAADF89460A76F50F4DEB18D6F445A80113DF050B0A1D3C68C957AEBC
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/F70CF10F1405D4FCDC24D6AD6D37C3CE7030DD3CAADF89460A76F50F4DEB18D6F445A80113DF050B0A1D3C68C957AEBC
http://www.w3.org/2000/01/rdf-schema#comment
"these studies established a critical role of Jmjd3-mediated H3K27 demethylation in Th17 cell differentiation and suggest that Jmjd3 can be a novel therapeutic target for suppressing autoimmune responses."
xsd:string
http://purl.uniprot.org/uniprot/#_45581C9885A3F18A7D71D5A14F745D046021E3DBCC66F8063C2EE9ABB91ECCC11A51C93E38BBD9203869782815A5C591
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/F70CF10F1405D4FCDC24D6AD6D37C3CE7030DD3CAADF89460A76F50F4DEB18D6F445A80113DF050B0A1D3C68C957AEBC
http://purl.uniprot.org/uniprot/Q5NCY0
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F70CF10F1405D4FCDC24D6AD6D37C3CE7030DD3CAADF89460A76F50F4DEB18D6F445A80113DF050B0A1D3C68C957AEBC
http://purl.uniprot.org/uniprot/#_Q5NCY0-mappedCitation-25840993
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F70CF10F1405D4FCDC24D6AD6D37C3CE7030DD3CAADF89460A76F50F4DEB18D6F445A80113DF050B0A1D3C68C957AEBC