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DESCRIBE <http://purl.uniprot.org/SHA-384/F7DD6993241FBF8CAEF510209040F477F278D3FFEF85D0B9CDD8943858BA18D09F6F84D92B31A7867744ACC3A3F91A1B>
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http://purl.uniprot.org/SHA-384/F7DD6993241FBF8CAEF510209040F477F278D3FFEF85D0B9CDD8943858BA18D09F6F84D92B31A7867744ACC3A3F91A1B
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/F7DD6993241FBF8CAEF510209040F477F278D3FFEF85D0B9CDD8943858BA18D09F6F84D92B31A7867744ACC3A3F91A1B
http://www.w3.org/2000/01/rdf-schema#comment
"Genetic variants of CYP21A2 associated to autoimmune Addison's disease(AAD) are in linkage disequilibrium with the main AAD risk locus HLA-DRB1 and CYP21A2 does not constitute an independent susceptibility locus."
xsd:string
http://purl.uniprot.org/uniprot/#_92F6E2315E2D198AD10C65DB900DA684D339CAF5F87E8C4DD371900F2227994764082BBAF0123251A438AA6FB0C16134
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/F7DD6993241FBF8CAEF510209040F477F278D3FFEF85D0B9CDD8943858BA18D09F6F84D92B31A7867744ACC3A3F91A1B
http://purl.uniprot.org/uniprot/F8RHL0
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F7DD6993241FBF8CAEF510209040F477F278D3FFEF85D0B9CDD8943858BA18D09F6F84D92B31A7867744ACC3A3F91A1B
http://purl.uniprot.org/uniprot/#_F8RHL0-mappedCitation-25249698
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/F7DD6993241FBF8CAEF510209040F477F278D3FFEF85D0B9CDD8943858BA18D09F6F84D92B31A7867744ACC3A3F91A1B