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DESCRIBE <http://purl.uniprot.org/SHA-384/FA058A68D5F4EBDD640C338C623D909037F5F13E14CB5A4C10AB58F3BFF4DBB463FB02CE29B363CA520BBAB951A1AE7E>
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http://purl.uniprot.org/SHA-384/FA058A68D5F4EBDD640C338C623D909037F5F13E14CB5A4C10AB58F3BFF4DBB463FB02CE29B363CA520BBAB951A1AE7E
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/FA058A68D5F4EBDD640C338C623D909037F5F13E14CB5A4C10AB58F3BFF4DBB463FB02CE29B363CA520BBAB951A1AE7E
http://www.w3.org/2000/01/rdf-schema#comment
"most severe phenotypes appreciated by the duration of transplant-free survival were caused by ABCB4 variants that were markedly retained in the endoplasmic reticulum and expressed in a homozygous status"
xsd:string
http://purl.uniprot.org/uniprot/#_14BA82424A444D1F97198DB04E0991F406D1D60958913BAD1775DB994E80B96D1C231156DDD9D8ACEF167C9D82EFFCA1
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/FA058A68D5F4EBDD640C338C623D909037F5F13E14CB5A4C10AB58F3BFF4DBB463FB02CE29B363CA520BBAB951A1AE7E
http://purl.uniprot.org/uniprot/E5LR39
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/FA058A68D5F4EBDD640C338C623D909037F5F13E14CB5A4C10AB58F3BFF4DBB463FB02CE29B363CA520BBAB951A1AE7E
http://purl.uniprot.org/uniprot/#_E5LR39-mappedCitation-26474921
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/FA058A68D5F4EBDD640C338C623D909037F5F13E14CB5A4C10AB58F3BFF4DBB463FB02CE29B363CA520BBAB951A1AE7E